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Mechanistic modeling of the MAT2ASRFPARN axis reveals a SAM-independent pathway that stabilizes SRF, drives glycolysis through PI3K/AKT activation, and promotes osteosarcoma progression, highlighting MAT2A as a therapeutic target.
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Graphical Abstract Schematic illustration of the proposed molecular mechanism highlights the role of dihydromyricetin (DHM) in the pathophysiology of SCI and subsequent motor function recovery
