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[4] The proposed mechanism of action, based only on preclinical animal studies, involves potential modulation of growth factor pathways, particularly vascular endothelial growth factor (VEGF), which may promote angiogenesisthe formation of new blood vessels

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[2] A study of chicken embryos demonstrated that BPC-157 increases vascular growth through the activation of cell surface receptors in the Nitrous-Oxide pathway, growing new vessels toward the injury

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6B and D), demonstrating that the AR-AST System can accurately identify bacterial resistance