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2c), most cells in GCGR-deficient mice were mTOR and p-STAT3-S727 positive and accompanied by elevated VGF levels and hypersecretion of glucagon (Fig

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Also, an activation mechanism involving an attack of H 2 S/HS at a disulfide in the intracellular kinase core was postulated for EGFR regulation ( + /K + -ATPase endocytosis in renal tubular epithelial cells through a downstream signaling pathway