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Proteasomal Degradation of the Histone Acetyl Transferase P300 Contributes to Beta-Cell Injury in a Diabetes Environment

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Feb 2021;53(1):e13644

The worsening of symptoms after stopping glutathione reflects the return of the underlying condition elevated oxidative stress from chronic disease, mitochondrial dysfunction, or metabolic pathology that glutathione was temporarily mitigating

Central to this dysfunction is impaired mitochondrial oxidative phosphorylation, which significantly reduces adenosine triphosphate (ATP) production, consequently impairing cardiac contractility 4,5,6