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This level of personalization is especially beneficial for chronic conditions like diabetes, where ongoing adjustments to treatment may be necessary

resistant alleles at low frequencies) and investigate the underlying molecular mechanisms driving this resistance (i.e

The ferroptosis inhibitors ferrostatin-1 (Fer-1), liproxstatin-1 (Lip-1), iron chelator deferoxamine (DFO) and the antioxidants NAC and Trolox 8,28 rescued the lethal erastin, while the apoptosis inhibitor Z-VAD-FMK, 29 and the necroptosis inhibitors necrosulfonamide (NSA), 7-Cl-O-Nec1 (Nec-1s) 28 failed to block erastin-induced death (Fig

DOI:10.1186/s12929-024-01093-8 32