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This proliferative capacity in the absence of resistance-conferring mutations is thought to reflect stabilization of adaptive non-genetic programs, including persistent epigenetic remodeling, transcriptional and translational rewiring, and metabolic adaptation 11,38,58,59 , which together enable limited growth without reactivation of full oncogenic signaling

Diabetes 55: 29392949, 2006 [DOI] [PubMed] [Google Scholar] 79.Park J, Chung JJ, Kim JB

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