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m6A-YTHDF1-mediated TRIM29 upregulation facilitates the stem cell-like phenotype of cisplatin-resistant ovarian cancer cells

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To view a copy of this licence, visit About this article Cite this article Hitzler, S.U.J., Fernndez-Fernndez, C., Gnther, K

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These findings illuminate the functional consequences of disconnecting histone modification crosstalk for a key epigenetic enzyme

This is consistent with many animal studies and in-depth immunological studies that show, as expected, a measurable contribution to protection via non-nAb [56,57,58], NK cells [59], and MAIT cells [60, 61] and in particular T cells [62,63,64,65]