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doi: 10.1016/j.brainresbull.2018.11.025
For example, in alcoholics, pools of mitochondrial GSH are depleted with the concomitant result that ROS damage can be exacerbated producing cell death and contributing to cirrhosis
Structural insights into the mechanism of the sodium/iodide symporter
Finally, the amount of this flexible pool of reduced and glutathionylated GRXs might be independent from local perturbations of the cellular GSH/GSSG redox potential
Selenium regulation of the selenoprotein and nonselenoprotein transcriptomes in rodents
A., Rasheed R., Saleem M
