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Ng et al., Journal of Endocrinology observed targeted reductions in fat deposits without adverse metabolic effects
The level of intracellular glutathione is a key regulator for the induction of stress-activated signal transduction pathways including jun N-terminal protein kinases and p38 kinase by alkylating agents
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Concentrated in neuronal membrane microdomains known as lipid rafts, cholesterol modulates protein distribution and synaptic signaling
