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The hypothesis that these reactive intermediates are responsible for cell death is supported by the observation that a cell line selected for erastin resistance showed several-hundred-fold upregulation of AKR1C family genes (Dixon et al., 2014), which encode aldoketoreductases that, among other functions, reduce reactive end-products of lipid peroxidation to unreactive compounds (MacLeod et al., 2009)

doi: 10.1007/s11901-018-0390-1, PMID: [DOI] [PMC free article] [PubMed] [Google Scholar] 36.Bruyneel M, Serst T

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