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doi: 10.1161/01.atv.19.10.23

436 In NSCLC, the complementary roles of the TRX and GSH antioxidant systems are also evident, as simultaneous inhibitionvia Auranofin treatment combined with GSR (GSH pathway) losssynergistically elevates ROS levels, overwhelms cellular redox defenses, and triggers apoptosis, thereby sensitizing cancer cells to TRXR inhibition

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As long as cellular glutathione is above a level considered to be adequate, which is called homeostasis, GCL is inhibited from making Glyteine, no matter how much cysteine is available