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In COPD, neutrophil function is dysregulated: CS impairs neutrophil apoptosis, prolonging their survival and inflammation
Oxidation of C18 Hydroxy-Polyunsaturated Fatty Acids to Epoxide or Ketone by Catalase-Related Hemoproteins Activated with Iodosylbenzene
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Under oxidative stress, however, ROS accumulation induces intestinal cellular DNA damage and mitochondrial dysfunction, impairing goblet cell viability through apoptotic pathway activation
