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The two-hit vascular hypothesis of Alzheimer's disease proposes that BBB dysfunction allows neurotoxins to enter the brain parenchyma, which in turn leads to neuroinflammation[43]

GSH manifests in two forms, mercaptan reduction (GSH) and disulfide oxidation (GSSG)

In hippocampal tissues, mRNA expression studies at 1 h and 24 h, and strongly elevated ( Egr1, Akt1, Kras, Src, Foxo, Srf, Vegfr2, Nos3, Nos1 ) and decreased ( Nos2, Nfkb ) gene expression ( Mapk1 not activated) also support BPC 157 beneficial effect on brain lesions, given in reperfusion in stroke-rats [30]

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BloodSpot: A database of gene expression profiles and transcriptional programs for healthy and malignant haematopoiesis