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Additionally, it was shown that UPF1 is not degraded by -glutamyltranspeptidase, which performs glutathione breakdown

separation occurs in the terminal ileum in the presence of calcium, and Vitamin B12 enters the mucosal cell for absorption

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1 Introduction Alzheimers disease (AD) is a neurodegenerative disorder characterized by progressive cognitive decline, memory loss, and neuropsychiatric symptoms, representing one of the most critical global public health challenges of the 21st century ( In this context, ferroptosisa newly defined, regulated cell death modality driven by iron-dependent lipid peroxidationhas rapidly emerged as a frontier and focus in AD research ( Ferroptosis, a regulated form of cell death, plays a significant role in various pathological conditions, including neurodegeneration, cancer, and ischemiareperfusion injury ( Figure 1 ) ( Figure 1 This review aims to systematically elucidate the central role of iron dyshomeostasis and the ferroptosis process it drives in the pathogenesis of Alzheimers disease

Table 1 Clinical features and precision treatment strategies for subtypes of endometriosis-associated infertility
