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The success of carnitine therapy in ischemic heart disease was first attributed to the higher in fat oxidation in cardiac myocytes, which resulted in an increased energy supply

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Pharmacologically- or antibody-mediated inhibition of choline uptake favors diacylglycerol (DAG) accumulation and protein kinase C activation, resulting in altered cytokine secretion in response to LPS (70) ( Figure 3 )

The underlying pathophysiology is mostly unresolved, with immunological factors often, but not exclusively, occurring after an initial infection with a distinct or gradual onset

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