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Description
As shown in Figures 4AC, all the compounds that reduced H 2 S production in HepG2 cells (Figure 3) also decreased mRNA levels of CBS , CSE , and NQO1 genes, after 24 h of treatment
These byproducts contribute to membrane disruption and may facilitate ferroptosis, depending on the local oxidative environment and PUFA composition (65, 66)
The role of glutamate homeostasis in the pathophysiology of SUD and its implications for pharmacotherapy
About this article Cite this article Kumar, A., Palfrey, H.A., Pathak, R
2019;68:s1s106
Effects of amino acid transport limitations on cultured hepatocytes