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Description
Similarly, paclitaxel binds with -tubulin, the main component of the mitochondrial membrane, and opens the mitochondrial permeability transition pore (mPTP), resulting in a rapid mitochondrial depolarization and cytochrome c release from mitochondria in a neuroblastoma cell line ( in vitro studies, paclitaxel-induced mitochondrial dysfunction significantly hampered cellular respiratory function and ATP production in DRG neurons (Ikegami and Koike, 2003

European Journal of Drug Metabolism and Pharmacokinetics

DNA methylation readers in plants
also called PSAT1 alpha: PSAT1)

C.CisnerosA

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