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Interestingly, although previous evidence has suggested that AD patients generally demonstrate progressive deterioration in brain perfusion and clinical symptoms, a recent SPECT study depicted an increase in the perfusion of the precuneus in AD patients after the administration of ALC, along with a lack of further deterioration of cognitive and neuropsychiatric symptoms [52]

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Interestingly ApoC-II-mediated activation did not increase LPL activity in HR or PHP, which may reflect the structure of the TG analog substrate, the relative composition of the biological sample, and the presence of endogenous ApoC-II (data not shown)

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- Reduced lactic acid buildup to delay fatigue