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Patient-derived xenograft (PDX) cells that survived after treatment with various anticancer drugs showed lower risk scores (RS), lower KRAS expression levels, lower activation states of the RAS-MAPK signaling pathway, and significantly different expressions of ion channel transport genes, which may be related to drug resistance mechanisms (Kim et al., 2015)

recA gene The recA gene encodes the RecA protein, a highly conserved recombinase essential for DNA damage repair, homologous recombination, and the preservation of genome integrity (Table 2)

Springer US, Boston, MA (2002)

doi: 10.1016/j.jphotobiol.2018.08.014

Oxidative stress and vascular damage are considered two major cellular factors that lead to alterations in the TM