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& Altmeyer, M

Furthermore, HSP70 could be considered a potential marker to evaluate disease activity.3,5,6 On the other hand, MxA protein (human mixovirus resistance protein 1), inducible by IFN-, shows strong expression in active perilesional vitiligo skin, suggesting a contributing effect of IFN- in disease progression.7 Similarly, S100B is another DAMP released by damaged melanocytes, whose levels are increased in active vitiligo and can stimulate inflammatory responses.8 Also, high-mobility group box 1 (HMGB1) proteins can induce the production of chemokine ligands, such as CXCL1 or IL-8 by keratinocytes, acting in the recruitment of immune cells.9 Calreticulin (CRT) is another of the most studied molecules in vitiligo, as it induces melanocyte apoptosis and the release of membrane degradation products important for immunogenicity.10 Melanocyte adhesion deficiency Several research groups have demonstrated that melanocytes in vitiligo exhibit reduced adhesive properties

So you don't make assumptions and don't make anecdotal claims, because when multiple factors change simultaneously, we have to have a more systematic approach, and we're all about systems, we're all about analysis and there are still ways that you can kind of tease out the data

Anti-inflammatory and profibrinolytic effect of insulin in acute ST-segment-elevation myocardial infarction

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The importance of nutritional aspects in the assessment of inflammation and intestinal barrier in patients with inflammatory bowel disease