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This was also confirmed in a previous study where in the initial 24 h-period following hepatic I/R injury, the occurrence of AKI was attributed to intrarenal vasoconstriction resulting from splanchnic vasodilatation following portal hypertension 19

Notably, the DHB/3-AP matrix exhibited efficient lipid detection performance in both positive and negative ion modes

These residues accounted for 77%, 20%, and 3% of the identified phosphorylation sites, respectively

Somers-Edgar TJ, Taurin S, Larsen L, et al

Does peptide therapy replace standard PMOS treatment

Ferroptosis: a new insight for treatment of acute kidney injury