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Ferroptosis is a form of programmed cell death regulated by multiple cellular metabolic pathways, such as iron metabolism, redox homeostasis, and mitochondrial activity, and executed by phospholipid hydroperoxides (PLOOHs), a form of lipid-based reactive oxygen species (ROS)[1, 2, 13]

L.ZavalaE.et al (2017a)

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c-Met signaling in the development of tumorigenesis and chemoresistance: potential applications in pancreatic cancer

Accordingly, the release of spermatozoa through more frequent ejaculations might be a potential strategy to attenuate the detrimental effects of ROS and improve perm quality (Figure 2) (115, 118)