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Which upregulate genes for fat oxidation (CPT1, ACOX1) and mitochondrial biogenesis (PGC-1)

Fat metabolism & lipolysis The primary reason it is sought: studied in preclinical work as an hGH (176-191) fragment for stimulating fat breakdown

However, in another study reporting administration of NR or the CD38 inhibitor GSK987A to mdx mice for 20 weeks 267 , treated mice with the CD38 inhibitor showed increased NAD + levels in muscle tissue, but no improvement was found in muscle function after treatment with either the inhibitor or with NR

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concentrations greater than 60 M were not tested due to known cytotoxic effects in 3T3-L1 cells (see Figure 1)

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