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Idiopathic pulmonary fibrosis (IPF) is a prototype featuring extracellular matrix expansion and lung structure destruction with unknown causative factors

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5-HT2A receptors stimulate ACTH, corticosterone, oxytocin, renin, and prolactin release and activate hypothalamic CRF and oxytocin-expressing cells

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368,369,370 HSF2 and DLG2 can block the oligomerization of ASC by interfering with the interaction between ASC and NLRP3, thereby inhibiting the activation of the NLRP3 inflammasome and the upregulation of IL-1 expression and alleviating the inflammatory response

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Rosenfeld RG, Bakker B

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I couldnt resist commenting Hello very nice blog

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