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Novel antitumor therapeutic strategy using CD4(+) T cell-derived extracellular vesicles

10.1016/S2215-0366(17)30236-5 70

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Toxicity, acetaminophen: Background

Using B16F10 melanoma cells and a mouse model of breast cancer, the authors reported that tumors reconstructed TDLNs by disruption of the subcapsular sinus macrophage layer, which is a route for antigens to enter the germinal centers (GCs)

Further, MOTS-c enabled 17% of the old mice to enter the final running stage (highest speed), whereas none in the untreated group were successful (Fig