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p53 controls the mTOR pathway at multiple levels, including direct regulating signaling mechanisms, posttranscriptional regulation by miRNAs, and inhibiting autophagy through proteinprotein interaction [56]

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As the production of hydrogen peroxide molecule was not allowed to reach a high value during infection due to the proper working of the CAT enzyme, this phenomenon might be responsible for providing resistance to the cultivars against the disease

Induction of HO-1 expression by iron-containing porphyrin hemin and treatment with the CO donor CORM-A1 protects EAE mice from acute disease, showing improvement in clinical score and reduced incidence of disease infiltration by polymorphonuclear cells (123)

Overall, structural alterations found in SURF1 and other LS mutant organoids confirmed the dysregulation of NPCs function and morphogenesis as a relevant mechanism in the pathophysiology of LS