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This may directly or indirectly lead to cerebral dysfunction, e.g., by inducing reperfusion injury, lactate production, BBB disruption, cerebral edema, ROS-production, activation of the Nod-like receptor family pyrin domain-containing 3 (NLRP3)-inflammasome or other processes that induce glial reactivity and aggravate CNS inflammation (for details, see section Abnormal CBF)

By addressing both the molecular and the cellular aspects of proteostasis, these strategies offer promising avenues for the treatment of disorders associated with protein folding and dysproteostasis

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