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ASH25-0015422-02A2)
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This bioactivity stems from multi-target mechanisms: significant suppression of key pro-inflammatory factors (TNF-, IL-1, IL-6, and iNOS) synthesis/release, enhanced antioxidant capacity ( P 0.05) and showed no significant difference in hepatic TG accumulation versus controls, collectively confirming BSPs metabolic safety and minimal hepatotoxic potential at these doses (He et al., 2025)
The role of Pdcd4 in tumour suppression and protein translation
SK-Hep1 0 cells were partially resistant to RSL3 and only a small portion (ca
Oncogene 20 , 35803584 (2001)
