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The neurotrophic factors or GT agents promote neuronal survival and repair, which offers potential disease-modifying effects (124)

Mitochondrial dysfunction can lead to the release of apoptogenic factors, while the activation of apoptotic pathways can exacerbate mitochondrial damage (Belhadj Slimen et al

Therapeutic Effects of Minocycline on Oleic AcidInduced Acute Respiratory Distress Syndrome (ARDS) in Rats. NaunynSchmiedeberg's Archives of Pharmacology 396, no

Neuroprotective effect of the natural iron chelator, phytic acid in a cell culture model of Parkinsons disease

Recently, it was reported that bile acid (a regulator of glycolipid metabolism) inhibits inflammasome activation by binding to transmembrane G protein-coupled receptor 5 on KCs and upregulates the production of anti-inflammatory cytokines ( Figure 2 )

Nitric oxide and neuronal death