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470 Recently, studies have shown that the GLP-1RA Meg peptide also enters the ejection fraction reserve following heart failure therapy

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For instance, metformin, a drug used to treat hyperglycemia, triggers AMPK activation through the AMPK-PGC1 pathway in human umbilical vein endothelial cells (HUVECs), leading to reduced mitochondrial ROS levels, the induction of SOD2, and the enhancement of mitochondrial biogenesis, effectively countering hyperglycemia-induced oxidative stress [153]