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While H 2 O 2 acts as a physiological signaling molecule, its excess generates hydroxyl radicals (OH) via Fenton chemistry, causing irreversible damage to proteins, lipids, and DNA

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L-2-oxothiazolidine-4-carboxylic acid (OTC) treatment attenuated plasma and tissue P-selectin levels, suggesting that the upregulation of P-selectin in hyperhomocysteinemic mice is also regulated by redox mechanisms [61]