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It is believed to be due to autoimmune induced damage to the thyroid causing excess hormone release, and for this reason is not responsive to antithyroid drugs, KI or KCLO 4 , but does, if treatment is necessary, respond to prednisone(67)

PMID 28137297

This resistance was limited to EGFR TKIs as PC9ER and PC9 cells were equally sensitive to conventional chemotherapeutic agents (Supplementary Figure S1B)

It requires an unbroken cold chain

H 2 S: a novel gasotransmitter that signals by sulfhydration

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