bpc 157 for tendon repair doses BPC-157 and the Difference Between an Evidence Gap and a Cover-Up: What the entire human evidence base actually looks like, and the questions to ask next. — WellFounded BPC-157: Tendon Repair and More

in preclinical models it activates the VEGFR2-Akt-eNOS signaling axis to promote angiogenesis and tissue vascularization ( It also engages ERK1/2 and FAK-paxillin pathways that regulate endothelial cell migration and fibroblast activity, and upregulates heat shock proteins (HSP70/90) for cytoprotective effects in gastrointestinal tissue models ( Research across multiple rodent injury models has characterized its activity in tendon, muscle, nerve, and GI tissue contexts

Injectable GHK-Cu reaches tissue concentrations roughly 1020 times higher than equivalent topical application

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Thus, regardless of some disputes [1], we can defend BPC 157 therapy for targeting angiogenesis and NOs cytotoxic and damaging actions but maintaining, promoting, or recovering their essential protective functions [3,4,5,6,7,8,9,10,11,12,13,14,16,17,18,19,20,21,22,23,24,27,28,29,30,31,32,34,37,39,40]

That complexity is the point

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