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Cholesterol homeostasis is controlled by two NCR proteins: NCR-1 absorbs cholesterol and promotes synapse development, whereas NCR-2 uses cholesterol and hinders synapse development

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Intriguingly, CEBPB also participates in HPV oncogene regulation: while it represses the HPV-18 long control region (LCR) via CEBPB, IL6/STAT3 signaling dominantly activates the LCR, highlighting a context-dependent interplay between CEBPB and viral oncogenesis [120]

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