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In mice with unilateral ureteral obstruction (UUO), miR-9-5p can prevent the downregulation of FAO-related genes, block TGF-1-induced bioenergetic disorders, induce reprogramming that affects metabolic disorders and mitochondrial dysfunction in renal tubular epithelial cells, and trigger a protective response against chronic kidney injury and RF [38]

The breakdown process appears to generate toxic intermediates or local concentrations of the proapoptotic domain that damage tubular cells

Beneficial properties of probiotics

Klein EA, et al

J Evid Based Dent Pract

However, whether ferroptotic cell death is triggered by A aggregation, or tau hyperphosphorylation or whether there is an interaction between them remains yet to be confirmed