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Further, a lower amplitude of compound action potential in the sural nerve suggested that the functional capacity of the heavily myelinated axons was preferentially compromised, leading to loss of peripheral sensation

Keywords : ferroptosis, diabetic kidney disease (DKD), regulators, molecular mechanisms, treatment progress In recent years, the number of individuals with diabetes kidney disease (DKD) is growing as the prevalence of diabetes mellitus (DM) has increased[1]

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TME is a complex ecosystem composed of tumor cells, extracellular matrix (ECM), innate and adaptive immune cells, inflammatory cells, tumor-associated macrophages (TAMs), cancer-associated fibroblasts (CAFs), tumor-associated neutrophils, myeloid-derived suppressor cells (MDSCs), endothelial cells, microvascular networks, as well as various cytokines and chemokines (Wu et al., 2022)