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In activated macrophage cultures, GHK-Cu administration reduces the production of pro-inflammatory cytokines including interleukin-1, tumor necrosis factor-, and interleukin-6 by 40-65% while simultaneously increasing the production of anti-inflammatory mediators such as interleukin-10 and transforming growth factor-

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Its mechanism of action is based on binding LPS as well as IL-1 and TNF-, which are components of the cell wall of Gram-negative bacteria, leading to their release from the bacterial wall, destruction of the cell wall, resulting in the death and destruction of the microbial cell