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In summary, S1PR2 adds another layer of complexity to BA signaling within the TIME, potentially acting as a receptor for conjugated BAs to influence cancer cell invasiveness and inflammation, possibly contributing to a pro-tumorigenic microenvironment in certain settings

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NOS3 variants that reduce eNOS activity or expression add a compounding layer: hypertension, coronary vasospasm, coronary artery disease, stroke, and preeclampsia are all associated with reduced eNOS function and impaired NO bioavailability