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These findings correspond to an apparent ~16-fold elevation over baseline GH exposure and more than a twofold increase compared with GHRP-2 alone

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Conclusion Tesamorelin and MK-677 both activate the GH/IGF-1 axis in preclinical and investigational research contexts but via fundamentally different upstream mechanisms: GHRH receptor activation (tesamorelin) vs GHSR-1a agonism (MK-677)

Roberts BR, Ryan TM, Bush AI, Masters CL, Duce JA

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