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Exogenous estrogen increased GCL, GS expression and increased GSH levels in livers of both male and female mice, but it had no effect in other tissues such as heart and brain (Liu et al., 2004)

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19,25,26 ), also produce hemophagocytic syndromes 27,28 , indicating that FHL3 pathology reflects impaired lytic granule exocytosis rather than disrupted late endosomal maturation 17

11 This in turn limits the risk of tophaceous gout progression and progressive joint damage, as well as preserving physical function and improving quality of life

Thus, QUIN is also a pro-oxidant compound, potentially neurotoxic, since its accumulation inside the CNS can result in hyperexcitation, increase of reactive oxygen species (ROS), and apoptosis (35)

doi: 10.1016/J.PNPBP.2020.110145