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In the protein backbone, TMAO contributes to the maintenance of functional conformations, enhancing stability without interfering directly with the protein's active sites, thus preserving activity [6, 7]

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During this activation, AECs can secrete several proinflammatory cytokines, such as tumor necrosis factor-alpha (TNF-a) 2,3 and Monocyte chemotactic protein-1 (MCP-1)

S-nitrosylation in Parkinsons disease and related neurodegenerative disorders

It has gained attention in research communities due to its unique structure and broad interest in experimental studies involving tissue pathways, recovery models, and peptide interactions

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