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This may be dependent on combined S1P1R and S1P2R signaling and the activation of the immediate early transcription factors ERG1 and AP1, indicating that astrocyte-derived HB-EGF is part of a rapid response mechanism that counteracts pro-inflammatory astrocyte functions (93)

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These proteins have subunits, five in mammals RelA (p65), RelB, p50 (from p105 precursor), p52 (p100 precursor), and c-Rel, which make dimers, but the most studied so far and the most widely expressed in cells is the p50/RelA dimer, NF-B

356 Since Tau can bind and stabilize actin, when Tau expression was increased, the over-stabilized actin prevented Drp1 from localization to mitochondria, down-regulated mitochondrial fission, lengthened mitochondrial morphology in neurons, and overproduced ROS, which induced neuropathy

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Taken together, these findings highlight how lipid dysregulation may be a unifying mechanism linking high UPF intake to pathophysiological changes underpinning anxiety