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Survivors with CRF often exhibit elevated levels of pro-inflammatory cytokines (e.g

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H3K18la directly activates pore membrane protein 121 (POM121) transcription, which enhances MYC nuclear transport and direct binding to the CD274 promoter to induce PD-L1 expression in NSCLC cells, thereby causing CD8 + T-cell dysfunction and promoting immune evasion of NSCLC

This is reported context, not a recommendation