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2 IMPLICATIONS IN CANCER Dysregulated MET signaling in cancer occurs through a variety of genetic or epigenetic mechanisms

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Research interest in BPC157 comes from its ability to help evaluate pathway behavior, receptor or target response, formulation stability, and analytical performance under controlled laboratory conditions

Furthermore, the model can be extended, in particular by using multi-omics approaches, such as metabolomics of the bacterial consortium and transcriptomics of the respective immune cells, providing mechanistic insights into the chemical-microbiome-immune interaction triangle