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The ability of S-Acetyl-L-glutathione to be directly absorbed by cells and converted to GSH without needing breakdown into amino acids presents a significant advantage, especially in conditions where GSH synthesis may be impaired, such as during viral infections or in genetic disorders affecting GSH metabolism

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We also demonstrated that NRF2 was upregulated in MT3 knockdown, with elevated Zn 2+ in preosteoclasts, and inhibition of intracellular Zn 2+ by TPEN decreased NRF2 expression

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