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Another interesting compound, quercetin, has been shown to inhibit the activity of CD38 by binding to its active site, which results in increased intracellular NAD + 143
doi: 10.59566/IJBS.2006.2143
Although Bcr-Abl inhibitors such as imatinib, nilotinib, and dasatinib are effective in CML therapy 7,8,9 , the acquisition of the T315I mutation of BCR-ABL in patients causes the resistance to most tyrosine kinase inhibitors (TKI), leading to poor outcomes
Domains for RCT studies were rated as low risk (+) when methods were clearly adequate, high risk () when methods were absent or likely to introduce bias, and unclear risk (?) when insufficient detail was reported (Table 5)
