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[DOI] [PubMed] [Google Scholar] 50.Hwang DF, Wang LC

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Elevated ROS levels disrupt cellular organelles, induce stress response pathways, and activate apoptosis and fibrotic signaling, collectively impeding effective tendon healing ( FIGURE 1 Understanding the sources and regulation of ROS in tendons, including the roles of nicotinamide adenine dinucleotide phosphate (NADPH) oxidases, mitochondria, and the hypoxia-inducible factor (HIF), is crucial for distinguishing between their beneficial and harmful effects