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2011 March 2

(1992) Site-directed mutagenesis of amino acid residues involved in the glutathione binding of human glutathione 5-transferase Pl-1, J

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When the amino acid supplementation was changed to one with depleted phenylalanine and tyrosine levels, serum T4 and T3 levels gradually decreased

Microsatellite stable and unstable oncogenic pathways associated with KRAS and BRAF mutations and epigenetic alterations pass through benign tubular adenomas or polyps and further oncogenes colorectal epithelial cells [16]

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