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Generally, these occur by mechanisms conveniently categorized as phase I and phase II metabolic transformations, which act in a tightly integrated manner [1,2]

Shank3 encodes for multidomain scaffolding Shank family proteins critical for postsynaptic function of glutamatergic synapses

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Unfractionated heparin ameliorates lipopolysaccharide-induced lung inflammation by downregulating nuclear factor-B signaling pathway

IFN- alone influenced tyrosine metabolism, glycine-serine metabolism, phenylalanine-tyrosine metabolism, tryptophan metabolism, cysteine metabolism, and lysine degradation

Uptake of oxidized lipids by the scavenger receptor CD36 promotes lipid peroxidation and dysfunction in CD8(+) T cells in tumors