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Mechanistically, SELENOP promotes IR by inhibiting the phosphorylation of Acetyl-CoA carboxylase (ACC), a critical enzyme in lipid metabolism

Assessment of endoplasmic reticulum glutathione redox status is confounded by extensive ex vivo oxidation

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This would make for more efficient transfer reactions, minimize the production of reactive oxygen species and be similar to metabolons of metabolic pathway enzymes, for which there is some evidence